oXXytocin Biology and Psychology. The "Point? CHANGE" of genetic personalities. How it Works?

 Remember that GG is Blue Personality and AA is Orange Personality.  To help in remembering which color is associated with which genetic personality traits, one might think of “A” as being “Away” (Orange) and “G” as being “Gregarious” (Blue). While these two words represent only one small facet of personality attributes associated with the two colors (representing oxytocin level ranges), they are a good starting point to help you remember the overall differences. 

Aside from these two characteristics, to gain more understanding about the myriad of differences between the two oXXytocin level ranges, you can read more information written in the scholarly articles found at this link: 

Scholarly Articles

There are actually 3 genetic variances (AA, AG, GG) for oXXytocin, known as polymorphic allelic variations located at the primary Oxytocin site on chromosome 3p25.  If there are 3 genetic variances, why are there only 2 genetic colored personality types?

There are only 2 genetic colored personality types: Orange (AA) and Blue (GG), even though there are three polymorphic allelic variations coding for oxytocin levels because the third variation of oxytocin (AG) has attributes that present in a personality type very similar to that of Blue.

The phenotype (presentation) of AG personality, which would be referred to as Green, does not have it’s own assigned personality color because the one G allele overpowers the one A allele, thus pulling it to the increased Oxytocin level side of the spectrum/scale referred to as Blue personality.

(That stated, there are some personality differences between Blue (GG) and Green (AG) as a result of carrying the one G nucleotide or marker in comparison to carrying the two G nucleotides, but differences are minimal so for now, LIVIN4d focuses on two personality types).

To parallel this oxytocin personality trait genetic transmission to the transmission of biological disorders, oxytocin could be considered similar to a point “mutation” (change) but without the personality trait being considered a disease as it does not have deleterious manifestations in the phenotype presentation.  It will be referenced as a “Point Change” to eliminate the “mutation” stigma.

Examples of “point mutations” can be observed in Sickle Cell Anemia or Cystic Fibrosis. Sickle Cell Anemia is located on Chromosome 11 and Cystic Fibrosis is located on Chromosome 7.  Each condition requires only one changed letter on chromosome 11 or 7 for the disease to manifest, similar to the change of an A to a G or vice versa on chromosome 3, manifesting a high or low level oxytocin personality range within a person.

(On a side-note when we more closely examine Sickle Cell Anemia and Cystic Fibrosis, it can be seen that heterozygous genetic carriers of the conditions have genetic and phenotypic assets with respect to the carriers' environments. A Sickle Cell Anemia carrier has a natural immunity to malaria while having no or very little symptoms of sickled hemoglobin.  Individuals carrying Cystic Fibrosis would survive extreme dysentery while having no increased production of mucosal goblet cells.   Science demonstrates that there are many assets to heterozygosity for many genetic diseases, like a win win of both worlds: carriers have the benefit of the disease without having the expressed symptoms.  

In terms of the oxytocin scale, the “would be” green or AG personality type is the genetic heterozygous form of the high levels of oxytocin and the low levels of oxytocin (even though Green’s phenotype (observable traits) is more similar to that of a high level oxytocin scale person).  To parallel this oxytocin personality trait phenotypic transmission to the transmission of biological disorders, the G nucleotide for increased oxytocin could be considered similar to an autosomal dominant disorder in that only one copy of a G is needed for higher levels of oxytocin to occur within an individual.  To repeat, levels of oxytocin are not "disordered."  An analogy is simply being made to transmission of genetic and phenotype traits.

It might be argued that similar to being a carrier for Sickle Cell Anemia and/or Cystic Fibrosis, the “would be” Green personality type has a natural personality “immunity/survival” in that it can travel and relate to all ranges of oxytocin personalities on either side of the spectrum (even though its phenotype manifests more blue personality traits).  Perhaps “would be” Green transcends many limiting effects experienced by being an Orange or Blue. Perhaps "would be" Green is immune to "malaria or dysentery " of the psyche and perhaps we don't yet even know what those are in terms of psyche.

Why the heck does this matter?  Who cares?  

Well, I don't know about the ladder, but it matters because knowing what personality type you are provides you with social navigational precision.  It externalizes innate assets and liabilities such that you can strengthen parts of personality and soften more salients aspects.  It allows for perception and knowing of the self and other in new ways, thus leading to expansion of connection.

Why does the genetic science behind Oxytocin and the mode of transmission matter?

Because most psychological theories and psychological methodologies or ways of understanding of people are subjective.   Color coordinated personalities finally blends true science with psychology.  It's no longer a guesswork of labels.  It goes below the levels of conditioning:  race, gender, age, socioeconomic level.  LIVIN4d is "base"d in truth.  

Speaking of which, I'm sure various "DSM-5 and ICD-10" diagnoses are associated with Blue or Orange personality types.  Eliminate diagnostic bulleted criteria and/or convert "NOS" disorders or those Not Otherwise Specified into diagnosable conditions. . . .if you want.

LIVIN4d is not about pathology.  I'm just imagining the tangential potentials.